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Inventi Impact: Pulmonology

Articles

  • Inventi:hpm/31685/20
    Upregulation of Potassium Voltage-Gated Channel Subfamily J Member 2 Levels in the Lungs of Patients with Idiopathic Pulmonary Fibrosis
    Research 2020 : July - September
    Jong-Uk Lee, Hun Soo Chang, Chang An Jung, Ryun Hee Kim, Choon-Sik Park, Jong-Sook Park

    Background. Fibroblast dysfunction is the main pathogenic mechanism underpinning idiopathic pulmonary fibrosis (IPF).\nPotassium voltage-gated channel subfamily J member 2 (KCNJ2) plays critical roles in the proliferation of myofibroblasts and\nin the development of cardiac fibrosis. Objectives. This study aimed to evaluate the role of KCNJ2 in IPF. Methods. KCNJ2\nmRNA expression was measured using real-time PCR in fibroblasts from IPF patients and normal controls (NCs). Protein\nconcentrations were measured by ELISA in bronchoalveolar lavage (BAL) fluid obtained from NCs.................

    How to Cite this Article
    Attribution/ CC Compliant Citation: Lee, Jong-Uk, et al.\n\"Upregulation of Potassium Voltage-Gated Channel Subfamily\nJ Member 2 Levels in the Lungs of Patients with Idiopathic\nPulmonary Fibrosis.\" Canadian respiratory journal 2020\n(2020).\nhttps://doi.org/10.1155/2020/3406530\nhttp://creativecommons.org/licenses/by/4.0/\n\nSome formatting elements, header, footer, logos, dates and\npagination were modified while adapting this article.
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